Thirty-four-year-old man without cardiovascular risk factors who was a semi-professional rugby player. He referred chest pain after thoracic trauma (TT) during a match; he presented with poor general condition, bruising, and sternocostal tenderness. In view of the poor evolution, electrocardiogram (ECG) was performed under analgesia (Fig. 1A); by virtue of the findings, urgent coronary angiography was requested, which revealed an anterior descending (AD) artery occlusive dissection (Fig. 1B), with intramural hematoma image on intravascular ultrasound (IVUS; Fig. 1C); the patient was treated with drug-eluting stents implantation (Fig. 1D).
High-energy closed TTs, including those observed in contact sports1, can cause serious injuries to internal organs. Although uncommon, traumatic coronary artery dissection is difficult to diagnose when clinical presentation, epidemiology, and patient characteristics do not reveal a coronary syndrome. The pathophysiology is largely unknown but, like other large vessels, traction-distraction forces appear to be the cause. There are no clear predisposing factors; according to some authors, collagen alterations, and non-significant atherosclerosis can modify the arterial structure and make it vulnerable.
Percutaneous revascularization is the strategy of choice in cases of occlusion or severe stenosis. In general, thrombolysis is not considered due to the differences in etiopathogenesis. Cases of conservative treatment have been described with good results in non-obstructive injuries, including surgical revascularization. Intracoronary imaging, although controversial, completes the diagnosis, and helps select treatment2.
Although there are centers with established protocols, it is safe to say that in every high-energy TT, or in those that are lighter but with incoercible pain or clinical involvement discordant with examination, necrosis markers’ serialized analysis, and ECG are mandatory.